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Indexado |
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DOI | 10.1186/S40659-017-0113-Z | ||||||
Año | 2017 | ||||||
Tipo | artículo de investigación |
Citas Totales
Autores Afiliación Chile
Instituciones Chile
% Participación
Internacional
Autores
Afiliación Extranjera
Instituciones
Extranjeras
Conclusions: BJ-1108 treatment ameliorates inflammation and EAE by inhibiting Th1 and Th17 cells differentiation. Our findings suggest that BJ-1108 is a promising novel therapeutic agent for the treatment of inflammation and autoimmune disease.
Ord. | Autor | Género | Institución - País |
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1 | Kang, Youra | - |
Yeungnam Univ - Corea del Sur
Yeungnam University - Corea del Sur |
2 | Timilshina, Maheshwor | - |
Yeungnam Univ - Corea del Sur
Yeungnam University - Corea del Sur |
3 | Nam, Tae Gyu | Hombre |
Hanyang Univ - Corea del Sur
Hanyang University - Corea del Sur |
4 | Jeong, Byeong Seon | - |
Yeungnam Univ - Corea del Sur
Yeungnam University - Corea del Sur |
5 | Chang, Jae Hoon | Hombre |
Yeungnam Univ - Corea del Sur
Yeungnam University - Corea del Sur |
Fuente |
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Ministry of Science |
Yeungnam University Research Grant |
Ministry of Science, ICT & Future Planning of the Korea government (MSIP) |
Basic Science Research Program from the National Research Foundation of Korea (NRF) |
Agradecimiento |
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This work was supported by the Basic Science Research Program from the National Research Foundation of Korea (NRF), funded by the Ministry of Science, ICT & Future Planning of the Korea government (MSIP) (2015R1C1A1A02036328, NRF-2014S1A2A2027903 and NRF-2014R1A4A1071040), and by a Yeungnam University Research Grant. |